Clenbuterol Bronchodilator Sopharma 50 tablets 0.02 mg

Clenbuterol Bronchodilator Sopharma 50 tablets 0.02 mg

SKU:294
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Bronchodilator, is a stimulant of beta2-adrenergic receptors of prolonged action. Bronchodilator effect due to direct relaxing effect on the muscles of the bronchi
Active substance:Clenbuterol
Pharmacological group:bronchodilating agent
Formulation:Tablets
Dosage mg:0.02
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$21
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Composition:
Clenbuterol hydrochloride 0.02 mg Excipients: lactose monohydrate 70 mg, wheat starch 31.48 mg, microcrystalline cellulose 48.5 mg, colloidal silicon dioxide 2 mg, magnesium stearate 2 mg, povidone (povidone K25) 6 mg.

Pharmacodynamics:
Bronchodilator, is a stimulant of beta2-adrenergic receptors of prolonged action. The bronchodilator effect is due to a direct relaxing effect on the muscles of the bronchi. May slightly increase heart rate. It also has a tocolytic effect.

Pharmacokinetics:
It has a high degree of absorption. High concentrations are created in plasma 15 minutes after a single dose and persist for 7-24 hours. It is excreted mainly by the kidneys. T1 / 2 - 3 hours

Side effects:
From the cardiovascular system: tachycardia, extrasystole, lowering blood pressure. From the side of the central nervous system: trembling fingers, anxiety, headache. From the digestive system: dry mouth, nausea. Allergic reactions: skin rash. Other: hypokalemia.

Features of the sale:
Prescription Special conditions The development of resistance and rebound syndrome is possible. Do not allow the drug to get into the eyes, especially with glaucoma.

Indications:
Prevention and relief of bronchospasm in bronchial asthma; bronchitis with bronchial obstructive syndrome.

Contraindications:
hyrotoxicosis, tachycardia, tachyarrhythmia, subaortic aortic stenosis, acute period of myocardial infarction, pregnancy, hypersensitivity to clenbuterol.

Drug Interactions:
Beta-blockers are pharmacological antagonists of Clenbuterol and can eliminate its effect. Clenbuterol reduces the effect of hypoglycemic agents. Increases the risk of developing intracardiac conduction disturbances while prescribing with MAO inhibitors and theophylline.

Increases cardiac glycoside toxicity and increases the risk of arrhythmias. In combination with sympathomimetic agents, a mutual increase in toxicity occurs.

Dosage:
Accepted orally in a dose of 2.5-20 mg 2 The dose is established depending on the patient's age and clinical situation

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